// App-Quantinova.ai

: SARS-CoV-2 infection drives endothelial dysfunction indirectly through hypoxia and increased pro-inflammatory cytokine production

Researchers

Presenter

  • Taejoon Won

Principal Investigators

  • Megan K Wood

  • David M Hughes

  • Monica V Talor

  • Roger A Johns

  • Nisha A Gilotra

  • Andrew S Pekosz

  • Jody E Hooper

  • Daniela Cihakova

Medical Centers

  • The Johns Hopkins University School of Medicine, MD.

  • Johns Hopkins University, MD.

  • Johns Hopkins Bloomberg School of Public Health, Baltimore, MD

Locations

  • United States

Companies

  • N/A

Study Components

Therapeutic Area

  • Pulmonary/Respiratory Diseases

  • Cardiovascular (CVS)

  • Blood/haematological Diseases

  • Infectious Diseases (ID)

Disease

  • Vasodilation dysfunction

  • Venous thrombosis

  • Coronavirus disease

Biomarkers

  • E-selectin

  • von Willebrand factor

  • Vascular cell adhesion molecule 1

  • Intercellular adhesion molecule 1

Drug/Treatment

  • Thrombomodulin alfa

  • ICAM-1

Outcome

  • N/A

Study Design

  • N/A

Phase

  • N/A

Study Id's

  • N/A

Sponsors

  • N/A

Result

  • N/A

Copyright © 2026. QuantiNova All rights reserved